hypertrophied

英 [haɪˈpɜːtrəʊfid]

美 [ˈhaɪpərˌtroʊfid]

网络  淋巴器官比正常时肥大; 肥大; 心肌肥大; 过度增大的

COCA.44733

英英释义

adj

• (of an organ or body part) excessively enlarged as a result of increased size in the constituent cells
  1. hypertrophied myocardial fibers
  Synonym:enlarged

双语例句

• The Acute and Chronic Effects of Amiodarone on Currents in Isolated Ventricular Myocytes of Hypertrophied Rats
  胺碘酮对大鼠肥厚心室肌细胞离子流的急性及慢性作用研究
• Conclusion: ADM is activated as a circulation hormone in thy hypertrophied model.
  结论：在此心肌肥厚模型中肾上腺髓质素作为循环激素被活化，可能是一种代偿性升高。
• The present study was to observe these two factors in the hypertrophied ventricles and aortas of sinoaortic-denervated rats.
  本研究观察去窦弓神经大鼠肥厚心室和主动脉组织中这两因子的改变。
• Aim Effects of captopril on angiotensin ⅱ type 1 receptors ( AT1R) and angiotensin ⅱ type 2 receptors ( AT2R) were studied in excess load-pressured hypertrophied cardiac muscles of adult rats.
  目的研究卡托普利对压力超负荷性肥厚心肌中血管紧张素Ⅱ1型受体与2型受体表达的影响。
• The I Kr and I Ks are reduced in hypertrophied cardiac muscle, which lead to prolongation of action potential duration and provide conditions for early after depolarization and TdP.
  心肌肥厚时IKr、IKs的减少导致动作电位时间（APD）延长，为早后除极（EAD）和TdP的产生创造了前提条件。
• In contrast it is characterised with multi-channels and nonspecific disorder in the hypertrophied myocardium in the acquired heart disease.
  而后天性心脏病的心肌肥大是多离子通道病变而非特殊性。
• Expression of Ang ⅱ receptors in excess load-pressured hypertrophied cardiac muscles of rats
  压力负荷性大鼠肥厚心肌中AngⅡ受体的表达
• [ Conclusion] Atorvastatin can prevent partly gap junction remodeling of hypertrophied myocardium induced by pressure overload.
  结论阿托伐他汀能部分防止压力负荷诱导的心肌肥厚时的Cx43重构。
• Effect of atorvastatin on gap junction remodeling in hypertrophied myocardium Propranolol pretreatment protects ischemic hypertrophied myocardium in rat
  普萘洛尔预处理对大鼠肥厚心肌缺血再灌注损伤的保护作用及其机制探讨阿托伐他汀对肥厚心肌缝隙连接重构的影响
• AIM To study the characteristic of the rapidly activating component ( Ikr) and the slowly activating component ( Iks) of the delayed rectifier K+ current in guinea pig hypertrophied ventricular myocytes induced by thyroxine.
  目的在甲状腺素诱导的豚鼠心肌肥厚模型上，研究肥厚心肌细胞延迟整流钾电流（IK）中快激活成份（Ikr）及慢激活成份（Iks）离子通道特征。